ResMed My Air program

So sorry to waste your and everyone's time. I can see that I simply do not have enough knowledge of the subject or understand the data that is made available my the machine I am using. I'll see if I can make an appointment with someone at the local center that supplied the machine to me who can possibly provide me with some understanding as to what I should be looking for and where to look for it. tnx

viewtopic/t172378/StickyIf-you-want-CPA ... Gquot.html

Please review the above thread.

To know how to maybe reduce the AHI we need to know what the event category breakdown of that AHI is.
How much is the
central event category
hyponea event category
obstructive apnea event category

Along with the usual question about "what are all your machine settings".

Reason to know each event category.....what we would suggest for the OA/hyponea category isn't what we would maybe suggest for central category.
Also need to know medications you are taking and are you sleeping soundly or waking often????

Here is what the Findings said. Maybe you can help me understand just what it says. thank you.


Sleep Architecture: Nocturnal polysomnography was notable for the decreased sleep efficiency at 52.0%. Sleep onset latency was prolonged at 611.2 minutes. Rapid eye movement sleep latency was reduced at 5.8 minutes. Sleep architecture was abnormal; stage N1 sleep was increased at 11.8%, stage N2 sleep increased at 61.1%, stage N3 sleep reduced at .02%, and stage REM sleep increased at 26.9%. Respiratory Parameters: During the night in the Sleep Medicine Center, the patient had a total of 60 apneas and hypopneas (6 central apneas, 8 obstructive apneas, 0 mixed apneas, and 46 hypopneas). The apnea-hypopnea index is 15.5 events/hr. The REM AHI is 10.6 events/hr. There were no respiratory effort related arousals; the respiratory disturbance index is 15.5 events/hr. There were45 spontaneous arousals for a spontaneous arousal index of 11.6 events/hr. Nocturnal oxyhemoglobin saturations reached a minimum of 78% in stage REM sleep. The patient spent 95.9 minutes of time in bed with an oxyhemoglobin saturation below 90%. The patient spent 21.1% of total sleep time in the supine position. EEG: EEG monitoring revealed no atypical findings. EKG: EKG monitoring revealed intermittent sinus bradycardia, intermittent sinus tachycardia, and occasional multifocal premature ventricular contractions. EMG;EMG monitoring revealed 3 periodic limb movements series for an index of 0.8% even'hr. There were 25 total limb movements for an index of 6.5 events/hr. There were no arousals associated with limb movements. Obstructive sleep apnea (G47.33), Comments: The nocturnal polysomnogram was notable for abnormal sleep architecture with evidence for sleep fragmentation and nocturnal hypoxemia due to obstructive sleep apnea. Sleep fragmentation can result in symptoms of disturbed nocturnal sleep and excessive daytime sleepiness; nocturnal hypoxemia can contribute to the pathophysiologic effects of sleep-disordered breathing. The patient did have evidence of some electrocardiographic abnormalities that were noted including sinus bradycardia, sinus tachycardia, and multifocal premature ventricular contractions. The patient may require further Cardiology evaluation if appropriate.

That's the diagnostic sleep study. Nothing in it answers any of the questions I asked.