Here's a repost to the link. Thanks Michael!
http://www.ucsfcme.com/2010/slides/MOT1 ... isease.pdf
I am basically just at my limit of understanding what's really happening here. Part of that is due, in part, to the effects of the centrals. I'm concerned that in writing to understand that what I've written might be seen as fact, when it's really more about wrapping my own head around it.
I've been on the same machine for several years. For the first bit, everything seemed to be improving. For my first night ever on the M-Series Pro, my AHI was 24. That came down over the course of a few days to 6. It took a couple months to figure out I needed to drop the cflex. That brought my AHI down to just over 2. Since then, it had been a case of titration t bring it generally below 2.
For years though, my AHI has hovered around 2, but I still wake up with massive headaches that don't go away during the day and brain fog that seems to be worsening. So I'm not sure it's a case of
resolving Complex Apnea. It seems to be getting worse. So I know I'm benefiting to some extent from CPAP therapy, but something is either missing, or I need a different type of xPAP therapy.
In three labs, they remarked that they were seeing centrals, but logged me as OSA. My airway doesn't close off. I'm pretty sure it never has.
It's entirely possible that I have Complex Apnea. Can you have Complex Apnea without the obstructive element?
SWS - filtering real data out of raw wave reports on what seems like a whim sounds not only professionally and scientifically malfeasant, but extremely difficult to attain, given the fluctuations in heart beat, the other rhythms you would have to screen out, and the fact that things like FOT show up at such high resolution. I'm not sure filtering just heartbeat from raw wave data is would be worth the effort in math-hours. Plus it's tampering with raw data. If anything, I would expect them to leave it as is to let others draw their own conclusions based on pure data rather than a patented process, just like seeing periodic breathing in the wave forms. Filtering seems improbable.
The problem with ramp is that recording is excluded from that period. If I can't record it, I can't use it. My pressure is so low, I sense that ramp is unnecessary anyway.
Thank you for the better explanation of hypopneas. Again, I'm still wrapping my head around it.
Here are some definitions I've learned since starting this S9 journey:
Eupnea - in the human respiratory system, eupnea (Greek eupnoia; from eu, well + pnoia, breath) is normal, good, unlaboured ventilation, sometimes known as quiet breathing or resting respiration. In eupnea, expiration employs only the elastic recoil of the lungs.
Bradypnea - Bradypnea (Greek from bradys, slow + pnoia, breath), British English spelling bradypnoea refers to an abnormally slow breathing rate. The rate at which bradypnea is diagnosed depends upon the age of the patient.
Dyspnea - Dyspnea or dyspnoea (pronounced disp-nee-ah, IPA /dɪsp'niə/), from Latin dyspnoea, from Greek dyspnoia from dyspnoos, shortness of breath), also called shortness of breath (SOB) or air hunger, is a debilitating symptom that is the experience of unpleasant or uncomfortable respiratory sensations. It is a common symptom of numerous medical disorders, particularly those involving the cardiovascular and respiratory systems; dyspnea on exertion is the most common presenting complaint for people with respiratory impairment.
Tachypnea - Tachypnea (or "tachypnoea") (Greek: "rapid breathing") is characterized by rapid breathing. It is not identical with hyperventilation - tachypnea may be necessary for a sufficient gas-exchange of the body, for example after exercise, in which case it is not hyperventilation. Tachypnea differs from hyperpnea in that tachypnea is rapid shallow breaths, while hyperpnea is rapid deep breaths. Tachypnea can also be a symptom of carbon monoxide poisoning in which oxygen delivery to the tissues and organs is blocked causing hypoxia and direct cellular injury.
Hypercapnia - Hypercapnia or hypercapnea (from the Greek hyper = "above" and kapnos = "smoke"), also known as hypercarbia, is a condition where there is too much carbon dioxide (CO2) in the blood. Carbon dioxide is a gaseous product of the body's metabolism and is normally expelled through the lungs. Hypercapnia normally triggers a reflex which increases breathing and access to oxygen, such as arousal and turning the head during sleep. A failure of this reflex can be fatal, as in sudden infant death syndrome. Hypercapnia is the opposite of hypocapnia.
Hypocapnia - Hypocapnia or hypocapnea also known as hypocarbia, sometimes incorrectly called acapnia, is a state of reduced carbon dioxide in the blood. Hypocapnia usually results from deep or rapid breathing, known as hyperventilation. Hypocapnia is the opposite of hypercapnia.
Psychopnea - Pretending to breathe when one isn't.
Pseudopnea - Pretending not to breath when one isn't.
Torocoprolipnea - the sense that one is breathing a huge load of BS. AKA called crapnea.
Okay, I made up the last three. Can you tell I took Latin in highschool?